Out of the 25 cases, Five of them were dissection with aneurysms and one was a case of Takayasu’s arteritis. Nine cases are of Stanford type A and have undergone surgical intervention, while one case deteriorated due to aortic rupture.

16 cases are of Stanford type B and based on the extent of involvement and stability of the patient are managed conservatively with medical treatment, endovascular repair or surgical intervention. 

Complications may be present at initial presentation in up to 30% of Acute aortic syndromes, and these have a profound effect on immediate management and intervention. Rupture and acute tamponade are the most consequential complications. At the root segment, an extension of a Stanford type A dissection or Acute aortic syndromes can compromise aortic valve function or tear through the annulus, causing immediate severe aortic regurgitation.

In the descending and abdominal aorta, complications include marked compression or obliteration of the true lumen, rupture, or continued growth of the false lumen with associated progression of dissection or rupture, Complications are primarily static or dynamic side-branch occlusion with resulting end-organ ischemia or infarct.